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FREE TESTING for watery mouth

FREE TESTING for watery mouth

Watery mouth is an ecoli infection of lambs that is often fatal.

During March 2019 APHA are offering FREE TESTING for watery mouth.

If you have lambs that have succumbed to this condition, please call the practice on 01772 861300 so we can arrange obtaining a sample for testing.

Foaling season

Exciting times if your mare is about to foal this season!

A couple of days before parturition the udder of the mare will develop wax tips on the end of the teats.

A normal birth usually takes about 30 minutes without any help. Directly after the foal is born the umbilicus is still attached. It is important to leave the mare and foal attached for as long as possible. The umbilicus will break at the right time and place, there is no need to cut it. The first couple of hours are important for the foal to drink enough colostrum. Colostrum is full of antibodies necessary for the immunity of the foal.

Within 8 hours the foals should drink approximately 2-3 litres of colostrum to get a sufficient enough antibodies.

The foal is only able to take up any antibodies in the first 24hours. If there are any concerns about the uptake of colostrum, a blood sample can be taken to test for IgG levels.

The placenta should be detached from the mare within 4 hours after parturition, it is important to check if the placenta is complete (needs to look like a pair of trousers). If the placenta has not come out within 4 hours, or is not complete it is important to contact the vet.

Did you know that a healthy foal should:

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  • Lift its head < 5 min

  • Stand < 2 hours

  • Drink < 2 hours

  • Urinate < 6-10 hours

  • Pass yellow feces < 24 hours (after dark meconium)

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It is advisable to have your mare and foal checked 24 hours after foaling, even if everything went smoothly. Foals are very good at fooling us! If the foal is a bit dull and quiet, not drinking enough or any other concerns you should ring your vet immediately. In these cases the earlier the foal is seen the better.

Injured cat found at local scrap yard

Two year old Bonnie was recently found with some serious injuries, under the bonnet of a scrap car at a local recycling plant.

Bonnie was brought to our Conway Drive surgery with a fractured tibia, broken canine tooth and some wounds around her face.

Unfortunately Bonnie is not microchipped, so local charity, Lancashire Cat Rescue, agreed to take her in.

The first stage of Bonnie’s treatment was for our vets to repair Bonnie’s leg with pins and an external frame to stabilise the fracture – see images.

The next stage planned is to remove Bonnie’s broken tooth when she has post op radiographs in 6 weeks time.

Bonnie is now recuperating with Lancashire Cat Rescue.

Diastemata

The 24 cheek teeth are designed to grind forage for up to 16 hours a day. These teeth erupt and are ground down continuously and those in each quadrant of the mouth act as a single grinding surface without spaces.

A diastema (plural, diastemata) is defined as a gap between teeth. Diastemata in the normally tightly fitting cheek teeth results in food entrapment. The stagnant food leads to painful periodontal disease (inflammation of the gums). If left untreated this can lead to the loosening of teeth, tooth root infections, sinusitis or food tracking up into the nose.

40-50% of horses suffer from diastemata. They are most common in older horses. It’s our most commonly treated dental disease.

Diastemata can result for many reasons:DIASTEMA

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  • With older age, teeth have erupted to the level of the slightly narrower root portion of the tooth.
  • Displaced (out of line) or rotated teeth- both are developmental defects, more common in breeds with small or dished faces e.g. miniatures.
  • Teeth drifting apart, towards the space remaining following a tooth extraction.
  • The teeth have erupted with diastemata between them, perhaps because the horse had grown bigger than the relative size of its teeth or erupted with out angulation.
  • Caries (decay) of the teeth which rots tooth away creating diastemata between them.

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Periodontal disease is very painful and horses often present with quidding (dropping of balls of hay). Some horses ‘pull faces’. Subtle signs e.g. eating less or weight loss are often attributed to old age. Symptoms are commonly seen during winter as long forage is more likely to become trapped between teeth than grass.

Diastemata are diagnosed at a routine oral exam, recommended every 6-12 months. To perform a thorough examination, a headtorch and mirror is essential; diastemata can be easily missed. Sedation is useful for fidgeting horses.

Treatment is carried out using sedation, pain relief and local anaesthetic in our clinic and involves removal of trapped food with the guidance of the oral endoscope camera that can view into the deepest pockets. Widening of diastemata is sometimes required to release entrapped food. The cleaned deeper pockets are sometimes packed with a protective putty whilst the gum heals beneath. Tooth overgrowths are floated (reduced) using powertools in order to balance the pressures on the teeth, reducing tooth drift. Severe cases may require tooth extractions.

Following a course of thoroughly performed treatment, the prognosis for resolution is very good especially with early diagnosis and action.

Sheep – The Big Five diseases

The Big Five Diseases in Sheep

Maedi Visna

Maedi Visna - sheep diseaseMaedi Visna is a wasting disease causing chronic pneumonia. It is caused by a highly contagious virus. Most sheep are infected as lambs via colostrum or aerosol, however generally do not show signs until they are 4 years old.

The main signs are:

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  • Thin ewes/wasting away
  • Pneumonia
  • Mastitis
  • Arthritis
  • Reduced fertility
  • Smaller weaker lambs which grow poorly due to a lack of milk

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Blood tests are commonly used to diagnose MV. Selecting thin cull ewes to test will help identify the disease if present within the flock.

If you suspect MV in your flock or wish to know your MV status we can do a 12 cull ewe screen.
Please call 01772 861300 to discuss.


Ovine Pulmonary Adenocarcinoma – OPA

Ovine Pulmonary Adenocarcinoma - OPA - sheep diseaseOvine Pulmonary Adenocarcinoma is caused by a virus leading to a progressive and fatal infectious lung cancer of sheep. The virus is spread from infected sheep by aerosol and nasal discharge. It is often seen in older thin sheep. There is no treatment.

The signs are very similar to chronic pneumonia:

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  • Difficulty breathing
  • Weight loss
  • Excessive watery fluid from the nose
  • Sudden death
  • Secondary infections such as Pasteurella are common.

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Once signs are evident the disease is fatal.

The definitive diagnosis is made by post mortem examination of the lungs.

If there is suspected OPA ultrasound examination of the lungs can be considered.


Johne’s Disease

Johne's disease in sheepJohne’s disease is caused by a bacterium which grows very slowly and lives for a long time in the environment. It is mainly spread through faecal contamination of feed and the environment, ewes also pass it to their lambs across the placenta and in colostrum. It is the same agent that causes Johne’s disease in cattle.

Infection generally occurs early in life, sheep often do not show signs for years and act as carriers, these are referred to as ‘sub-clinically infected’.

Affected sheep suffer from:

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  • Severe weight loss
  • Sudden death
  • Anaemia
  • High parasite burdens
  • Affected flocks suffer from poorer production, more thin ewes and increased culling.

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Testing for Johne’s can be tricky at an individual level. There is no single diagnostic test that can detect the disease at all the stages and sub-clinically infected animals may test negative.

Blood screening groups of cull/thin ewes can be effective at identifying Johne’s disease in your flock.


Border Disease

border disease in sheepBorder disease or hairy shaker disease is a virus which causes birth defects, barren ewes and abortion. It is from the same family of viruses as BVD in cattle.

If a ewe is infected during pregnancy the virus passes through the placenta to the lamb, in some cases this causes embryonic loss or abortion. Some lambs survive and are born Persistently infected (PI). PIs shed the virus continuously and can infect other sheep and lambs. This can be a major problem in naïve flocks unknowingly buying in PIs which can then cause around half of lambs born to be affected by the disease.

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  • Infection in non-pregnant adult sheep is generally short lived and mild, often no clinical signs are seen.
  • Hairy shakers are lambs infected with the virus whilst developing in-utero effecting their nervous system which leads to trembling and incoordination. They also have long curly wool.
  • Some lambs are born completely normal but maintain the infection, these are called Persistently Infected animals (PI).

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Hairy shakers are usually a good way to diagnose the presence of the disease. These lambs can also be blood tested to confirm presence of the virus.

Blood testing a group of ewes can identify if the flock has been exposed to the disease and is likely to be present.


Caseous Lymphadenitis – CLA

Caseous Lymphadenitis - CLA - sheep diseaseCLA is a chronic bacterial infection of the lymph nodes resulting in abscesses. The bacteria often enters via cuts and is commonly spread through close contact when animals are housed or are trough fed. The disease can also be transmitted indirectly such as on shearing equipment.

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  • Infected animals have lumps often behind the jaw, in the arm pit and in the groin. These lumps are often cold and painless but can burst open and drain pus. Once they have healed and scarred over the abscess can re-occur.
  • Abscesses also form inside the sheep causing ill-thrift and weight loss.

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Lumps and abscesses are a classic sign of the disease and should not be ignored.

Diagnosis is either by sampling the intact abscesses and culturing the pus or by blood test.


If you would like further information, please contact our Farm Team.

Lenny’s Tongue injury

Back in November, Lenny, a charismatic young dressage horse, was found hyper-salivating, unable to eat or open his mouth and very quiet when his owners did a routine late night check.

The emergency vet was called and Hattie attended to find a very unusual injury to Lenny’s tongue. The tongue had an almost full thickness jagged laceration to the top surface with a complete tear of the frenulum on the underside (the bit that connects the tongue to the floor of the mouth).

Injury on Hattie’s arrival

Torn frenulum (underside)

 

During the repair

Lenny underwent a general anaesthetic first thing in the morning to have the wound cleaned and repaired. Hattie was able to pull the wound together on the surface but left the underside to heal on its own due to the difficulty keeping the stitches in a mobile tongue.

3 days after repair

Lenny recovering

4 weeks after repair

Lenny made an excellent recovery thanks to his very caring owners and is now back in full work with no long term effects from his traumatic autumn evening!

Lenny during dressage

Neonatal Lamb Care

Neonatal Lamb CareLambing time is the most crucial part of the year for making your sheep business profitable. Lamb deaths from birth to 3 days old should be less than 7% however many farms range between 10- 25%.

A major factor influencing mortality in neonate (young) lambs is ewe body condition; as this directly relates to lamb body weight, colostrum quality and quantity. It is therefore important to ensure ewes are at BCS 2.5 to 3.0, (so vertebral spinous processes only felt with pressure and transverse processes only felt with hard pressure), and they are on a rising plane of nutrition in the run up to lambing. Energy and protein blood profiles can be taken 2 to 3 weeks before lambing to ensure ewes are receiving the correct nutrition to help reduce cases of twin lamb disease.

Another major cause of neonatal mortality is watery mouth. These lambs typically are lethargic, salivate and have a distended abdomen. It is caused by Ecoli and picked up by ingestion. Treatment involves electrolytes and antibiotics but is often hopeless. Therefore the emphasis should be on prevention which involves keeping pens clean, (especially towards the end of the season), dipping navels with strong iodine and ensuring all lambs get enough good quality colostrum quickly. See below:


Always ensure colostrum is…

QUALITY

Ensure ewes in good BCS, if you’re uncertain as to the quality of colostrum; colostrum quality can be measured using a refractometer: aim for specific gravity >1.05. Only use sheep colostrum replacement (colostrum from a milking sheep farm will usually be better quality than powdered colostrum); cow colostrum can cause a fatal anaemia. Also remember to give your Clostridial and Pasteurella vaccine four to six weeks before lambing to ensure the lambs receive antibodies against these diseases. (Any ewes that haven’t had this vaccine before will need a course of two injections four to six weeks apart).


QUANTITY

200ml/kg within 18 hours of birth, with a maximum of 50ml/kg intake on each occasion.

Remember lambs in adverse weather conditions will require more colostrum or milk as more energy from their feed will be used to keep them warm.


QUICKLY

Within 2 hours of birth.


SQUEAKY CLEAN

Ensure ewe udders are clean and dry and the equipment to mix and administer colostrum is cleaned and disinfected between lambs.


QUANTIFY

We can also look if your lambs have received enough colostrum by blood testing any less than a week old.


Finally, there is continual pressure on antibiotic use. So to ensure we don’t end up with a world full of super bugs we need to be careful how we use antibiotics. Preventative use of antibiotics, such as blanket use of Spectam or Betamox LA for watery mouth needs to be reduced as this will actively select for resistance and there is pressure from Red Tractor Assurance to reduce this practice.

We recommend only using antibiotics….

    • Towards the end of the lambing season, when the sheds have a build up of Ecoli.
    • In triplets, when their ewe may have poorer quality and quantity of colostrum
  • In lambs from ewes in poorer condition, as the quality and quantity of colostrum will be poorer.

Also remember entropion, where the lamb’s eyelid is inverted causing the eye to be held shut, tear staining and damage to the eye. This is a heritable condition so affected lambs should not be kept as replacements, ewes should not be used to breed replacements and consider ram choice if a large percent of flock are affected.

If you have any questions regarding lamb care, please contact the Oakhill Farm Team.

Mastitis in ewes

Mastitis in ewes can be fatal and usually results in the end of her productive life.

The usual presentation is acute gangrenous mastitis (blue or black bag) usually caused by either Staphylococcus Aureus or Mannheimia Haemolytica. S. Aureus is normally present on the teat skin but damage to the teat ends allows it access into the teat canal and causes mastitis.

M. Haemolytica is present in the tonsils of lambs so colonises the udder during suckling. The bacteria produces toxins which prevent blood flow and cause the udder to turn blue/black hence the name. This often occurs around peak lactation (three to six weeks post-lambing) and the initial clinical signs may be as subtle as a ewe looking lame or a lamb bleating because it’s hungry. The disease then rapidly progresses.


The risk factors include:

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  • Under feeding ewes round lambing or ewes in poor condition, as lambs to these ewes will cross suckle or create teat lesions by over suckling.
  • Damaged teat end or Orf on teats
  • Lumps in the udder
  • Lambing inside (the longer the time spent inside the greater the risk of developing mastitis)
  • Dirty, wet pens and dirty hands when handling udder/ underneath of sheep
  • Harsh, cold weather

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Treatment is unlikely to save the udder but may save the ewe’s life:

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  • Remove lambs from the ewe and ideally artificially rear as rearing these lambs on another ewe may spread the mastitis
  • Antibiotic injectable AND non-steroidal anti-inflammatory such as Loxicom
  • A ewe that has had mastitis will be more likely to get fly strike so preventative fly treatment is essential.
  • There is a vaccine against S. Aureus available; VIMCO. This has to be given three and five weeks pre-lambing. Most of the trial work has been done in dairy ewes and it is licenced to reduce to the incidence of subclinical mastitis. Potentially this could also help to reduce the incidence and severity of black bag in your flock however at the moment there is limited evidence to prove this.

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If you have any questions regarding mastitis in your flock, please contact our Farm Vet Team.

Eyelid lacerations

Oakhill Vet Stuart was called promptly when Sully caught his eye on a wall. 

Sully’s eye was checked for an ulcer and the eyelid was sutured under sedation and local anaesthetic. Prompt suturing and great care from his owner, preventing him from disrupting the stitches, has lead to a great cosmetic result!

However, eyelid laceration can have many complications:

  • Failing to get the  edges of the wound meeting nicely when stitching together can cause the eyelid or lashes to scratch the outer covering of the eye, the cornea.

  • If the eyelids can’t move as normal then tears can’t spread across the eye.

  • There are glands in the margin of the eyelid that secrete a component of the tears. If these are damaged there is an alteration the tear consistency.

The above three complications leads to corneal ulceration and pain.

Tears drain via the nasolacrimal duct in the inner corner of the eye. If obstructed or damaged, tears accumulate and then tear down the face which can cause skin irritation. Stagnant tears can attract bacteria and lead to conjunctivitis

To give the best chance of eyelid lacerations healing well and preventing the above mentioned side effects, veterinary assessment and suturing of wounds needs to occur promptly.